Characteristics of a Normal 12-Lead EKG

Voltage Standardization

• A full standard calibration mark appears as two vertical lines at the beginning or end of a lead, measuring 1 mV in amplitude or 10 mm tall (or two large squares). It looks like 3/4 of a vertical rectangle traced on the paper that helps verify proper voltage scaling of the EKG.
• A half standard calibration mark (not pictured) is 0.5 mV in amplitude or 5 mm tall. Reduced voltage scaling, like 5 mm/mV, may be used if QRS complexes are extremely large.
• Always check the calibration marks to confirm proper voltage standardization before interpreting EKG amplitudes. Abnormal calibration can lead to misdiagnosis of conditions, such as low voltage or left ventricular hypertrophy.

Figure 9.1  Diagram of electrocardiogram paper.^[1]

Rhythm

• Regular sinus rhythm
• P waves present and upright in leads I, II, aVF, and V₄ – V₆
• P waves inverted in lead aVR

Rate

• Normal rage: 60 – 100 bpm

P wave

• Smooth, rounded shape
• Monophasic in lead II
• Biphasic in V₁
• Duration: <0.12 secs
• Amplitude: <2.0 mm

Figure 9.2  Normal P wave morphology in leads II and V₁.^[2]

PR interval

• Normal range: 0.12 – 0.20 secs

QRS complex

• Duration: <0.12 secs
• Narrow and sharp appearance
• Amplitude greater than P wave

ST segment

• Isoelectric (flat) baseline
  • No elevation or depression
• Usually horizontal or upsloping in all leads

T wave

• Upright and symmetric in most leads, except aVR and V₁
• Amplitude: <5 mm in limb leads (i.e., leads I, II, III, aVR, aVL, aVF)
• Typically deflects same direction as QRS complex
  • Note. Changes in T wave morphology can indicate various benign or pathologic myocardial conditions

QT interval

• Corrected QT interval (QTc): <0.44 secs in men or <0.46 secs in women
• A normal QT is less than half the preceding R-R interval

Electrical axis

• Normal QRS axis is between –30º and +90º

Voltage

• Normal R wave progression from V₁ to V₆
• No abnormally tall or deep waves across all leads

Intervals and segments

• Consistent PR, QRS, and QT intervals across all leads

Figure 9.3  This 12-lead EKG illustrates normal sinus rhythm (NSR). Each lead displays the characteristic P wave, QRS complex, and T wave, with a consistent and regular rhythm. The heart rate is within the normal range of 60–100 bpm. This EKG can serve as a reference for identifying normal electrical activity in the heart, providing a baseline for comparison with abnormal rhythms.^[3]

Steps to Interpret 12-Lead EKGs

1. Is the rhythm regular or irregular?

• • Determine if the rhythm is regular or irregular by examining the consistency of the R-R intervals
  • Look for patterns that might indicate atrial fibrillation, atrial flutter, or other arrhythmias

2. Measure the heart rate

• • Use the rapid estimation or 1500 method for regular rhythms
  • For irregular rhythms, calculate the heart rate range between the shortest and longest R-R intervals using the 1500 method

3. Evaluate the P waves

• • Check if there is a P wave before each QRS complex, indicating atrial depolarization
  • Assess the morphology and consistency of P waves across different leads

4. Examine the PR interval

• • Measure the PR interval from the beginning of the P wave to the start of the QRS complex
  • Deviations from the normal PR interval may indicate AV block or pre-excitation

5. Assess QRS duration

• • Measure the width of the QRS complex
  • Prolonged QRS duration may suggest a bundle branch block or that the rhythm is of ventricular origin

6. Identify extra or early beats

• • Look for premature atrial contractions (PACs), premature junctional contractions (PJCs), or premature ventricular contractions (PVCs)
  • Note any patterns, such as bigeminy or trigeminy, which could indicate underlying issues

7. Check R wave progression

• • Ensure normal R wave progression in the precordial leads (V₁–V₆), with increasing amplitude from V₁ to V₄
  • Poor R wave progression may suggest anterior myocardial infarction or other pathologies

8. Evaluate ST segments

• • Examine ST segments for depression or elevation, which can indicate ischemia or infarction
  • Compare ST segment to isoelectric baseline or TP interval line to determine the extent and significance of deviations

9. Look for abnormal Q waves

• • Identify any Q waves that are deeper than 25% of the height of the R wave and are wider than 0.04 secs
  • Abnormal Q waves may signify previous myocardial infarction if present in a set of contiguous leads (e.g., lateral leads: I, aVL, V₅, V₆)

10. Assess T waves

• • Check the size and shape of T waves; they should be upright in most leads and symmetrical
  • Inverted of peaked T waves can indicate ischemia, hyperkalemia, or other conditions

11. Identify U waves

• • Look for U waves following the T wave, most visible in leads V₂ and V₃
  • Prominent U waves may indicate hypokalemia or other electrolyte imbalances

12. Detect myocardial ischemia or infarct

• • Look for signs of ischemia (i.e., ST depression) or infarction (i.e., ST elevation)
  • Correlate findings with clinical presentation and history for accurate diagnosis

13. Summarize findings and name the EKG

• • Integrate all observations to name the EKG, considering rhythm, rate, intervals, and any abnormalities
  • Provide a concise interpretation that includes potential diagnoses and recommendations for further evaluation

Figure 9.4  An acute anterolateral ST elevation myocardial infarction (STEMI). Hyperacute T waves and ST segment elevation are present in V2–V4, poor R wave progression is apparent, along with deep Q waves shown in V1–V2. ST elevation is displayed in leads I, aVL, and V5 with reciprocal ST depression in lead III.^[4]

Figure 9.5  Example of poor R wave progression. Notice the R wave amplitude does not increase as expected from leads V₁ to V₄, indicating a possible anterior myocardial infarction or other pathological conditions affecting the anterior wall of the left ventricle. ^[5]

Figure 9.6  Example of ST segment elevation and reciprocal changes, which is indicative of an acute myocardial infarction (MI). This EKG was taken from a 66-year-old male was complaining of chest pain. Acute ST elevation MI (STEMI) in the lateral leads I, aVL, V5, and V6 with reciprocal ST depression in leads III, aVF, and in V1–V4. Q waves are also present in the lateral leads. The patient was quickly admitted to the cath lab. His coronary arteries, including the left circumflex, all were free of occlusive lesions. He had no coronary spasm during the procedure, but it was decided that spasm had been the cause of the EKG changes. His EKG reverted to normal. ^[6]